Biological Causes Of Bipolar Disorder Psychology Essay

Biological Causes Of Bipolar Disorder Psychology Essay Bipolar disorder is likely one of the most difficult diagnoses to receive in terms of living a normal life. As a psychological disorder, or more specifically, a mood disorder, its typical age of onset is in the early twenties, and its prevalence is between one and two percent worldwide. The disorder is characterized by an alternation between mania and depression, as well as poor impulse control, attention deficits, and impairments of verbal memory. Intensity of the manic state in a bipolar patient determines whether they are diagnosed with bipolar I or bipolar II. Those with classic, full-fledged mania have bipolar I, where as those with a less extensive version involving anxiety or irritability, sometimes referred to as hypomania, have bipolar II (Kalat, 2013). Causes of the illness are not always clear, but one or a combination of factors such as neurochemical factors, environmental factors, and genetics usually play a part in the development of bipolar disorder (Bressert, 2007). Genetics are an essential factor, as bipolar disorder is a very biologically based illness. Certain genes have been shown to increase the risk of developing bipolar II disorder, and also some genes associated with a predisposition to major depression predispose to bipolar disorder. However, no definitive relationships exist between these genes and bipolar disorder (Kalat, 2013). Various statistics have been obtained through research as far as chances of developing bipolar disorder based on its prevalence within a family. For example, the risk of the illness is between fifteen and twenty-five percent for those with a parent who has bipolar disorder. Furthermore, the risk for someone whose non-identical twin has the condition is twenty-five percent, which increases eightfold if they are identical twins (Bressert, 2007). Biological Causes Overview Here is an overview of many different factors that are biological in nature and have implications in bipolar disorder, some of which will be explored in further detail later. As a primarily biological illness, the improper functioning of neurotransmitters such as serotonin, norepinephrine, and likely many others has been identified as a cause (Bressert, 2007). Another interesting biological factor includes the brains increased use of glucose throughout a manic episode and its decreased use of glucose during depressive episodes (Kalat, 2013). Studies analyzing the reelin gene which helps in brain function and development have found that there is a link between the dysfunction of the reelin gene and psychiatric disorders, specifically schizophrenia, and more importantly for this paper, bipolar disorder (Ovadia, 2011). On a side note, there have also been studies which have found a gene that may provide some sort of protection against bipolar disorder. It is called GRIK4, and as a kainate-type ionotropic glutamate receptor is part of the glutamate neurotransmission process (Pickard, 2006). Another interesting indicator of bipolar disorder was found using sensory gating, and involves the P85 gating ratio (Patterson, 2009). Relating to the circadian rhythm aspect of the disorder and how disruption of this due to the disorder can be a cause of the manic and depressive states in bipolar patients, there have been some studies. The results of one study found an association between the NR1D1 and GSK3ÃŽÂ ² variants and differences in functioning of the expression of genes related to the circadian clock system (McCarthy, 2011). An association specific to rapid cycling bipolar disorder involves the gene variation of CRY2, a clock gene (Sjà ¶holm, 2010). A biological marker related to gluten sensitivity has been discovered which showed that those with bipolar disorder have increased levels of IgG antibodies to gliadin (Dickerson, 2011). Finally, a recent study looked into gene expression in  lymphoblastoid cells, which could be a possible biological marker of bipolar disorder (Kato, 2011). Overall, it seems that there are still many questions as far as biological causes of the disorder, though progress tow ards the specifics of those markers is definitely being made. Circadian Rhythm Factors The bodys natural circadian rhythm is affected in those with bipolar disorder. This has been discovered by McCarthy et al. (2011) through research into the effects of a certain treatment, lithium. It has been shown that lithium affects the function of circadian rhythm with the goal of treating the disorder. Further research conveys issues with functioning of the circadian rhythm that lithium seems to treat. According to one study, positive lithium response is predicted by the combination of variants within GSK3ÃŽÂ ² and NR1D1. This shows that in bipolar patients, lithium affects circadian clock genes, specifically NR1D1 and GSK3ÃŽÂ ², in an attempt to alter circadian rhythms. Also, a transcription of NR1D1, called Rev-ErbÃŽÂ ±, functions as a clock signaling molecule which is important as it is degraded by lithiums inhibition of GSK3ÃŽÂ ². Lithium also can lengthen the natural circadian rhythm and regulate the stability and turnover of proteins within the clock mechanism (McCarthy, 2011). Therefore, it is fairly clear that lithium affects the circadian rhythm of bipolar patients. This is interesting from a biological standpoint so that further research can be done into which clock genes dont function properly due to the disorder, or if their dysfunction is what causes the disorder. As Sjà ¶holm et al. (2010) found, rapid cycling in bipolar disorder relates to a specific circadian gene, called Chryptochrome 2 (CRY2), which is a main part of the pathway responsible for the bodys roughly twenty-four hour cycle. Analysis of single nucleotide polymorphisms (SNPs) for associations to bipolar I and II and bipolar with rapid cycling led to analysis of haplotypes only for the rapid cycling cases. Rapid cycling patients were focused on since their circadian rhythms were hypothesized to be more vulnerable. Haplotypes of CRY2 that indicated risk and protection were discovered in the study of bipolar patients. AAAC and AGGA were haplotypes found frequently in the rapid cycling bipolar patients, and GGAC was a haplotype found much less frequently in rapid cycling patients. This provided evidence for the conclusions that CRY2 is involved in bipolar disorder, and more specifically, which haplotypes lead to protection from or risk of the disorder (Sjà ¶holm, 2010). Assessment of more clock genes was completed by Yang et al. (2009), in which rhythmic expression patterns were examined and some interesting results were reached. Fibroblasts of bipolar patients and healthy controls were analyzed during the study. The amount of expression for half of the genes being studied, BMAL1, REVERBa and DBP, was less in the bipolar patients than the controls, in addition to the mRNA expression levels in two of the genes, DEC2 and DBP. Next, four kinases were examined for mRNA expression levels, and two of the four, GSK3a and GSK3b, were examined for phosphorylation and protein levels. Results were that in GSK3b, the level of phosphorylation was much less in bipolar patients than in the controls. Overall, while nothing is made absolutely clear by this study, there is substantial evidence that circadian genes in bipolar patients are expressed differently than those in mentally healthy individuals. This difference may contribute to dysregulation of other genes do wn the pathway, which in turn might explain some of the issues that bipolar patients have, relating to their circadian clock (Yang, 2009). Therefore, circadian rhythms are a main component in bipolar disorder, and the more that becomes understood, the better that aspect of the illness can be treated in the future. For now, it seems to be mainstream knowledge that maintaining a constant sleep schedule can help these symptoms of the disorder, but soon there may be better options for those that suffer with this frustrating disorder (Kalat, 2013). Neurological Factors Functional flaws in the neurological systems of bipolar disorder patients are implied in the emotional impairment of those with the illness, according to Phillips et al. (2008). Since regulating emotions is a vital part of being able to function in modern society, bipolar patients are posed with a particularly difficult dysregulation. Neuroscience examining youth development of emotion regulation in their neural systems is key, as advances made in this area could help target predisposed individuals and intervene to possibly prevent the development of bipolar disorder. Structural and functional abnormalities in those with the disorder may be valuable biomarkers with the potential of earlier detection and more successful treatment. This would be a very relevant and applicable result for bipolar disorder specifically, since it is often discovered once it is too late for a successful treatment plan, or misdiagnosed altogether (Phillips, 2008). Areas of the prefrontal cortex that are involved in control processes such as regulation of emotions, and decision making are stated by Phillips et al. (2008) and include the orbitofrontal cortex (OFC), dorsomedial prefrontal cortex (MdPFC), anterior cingulate gyrus (ACG), dorsolateral prefrontal cortex (DLPFC) and ventrolateral prefrontal cortex (VLPFC) (Phillips, 2008). Networks involving the orbitofrontal cortex and the dorsomedial prefrontal cortex allow for coordination of sensory integration and visceral control of prominent emotional information and also assist emotional behavior regulation. The amygdala is also connected to the orbitofrontal cortex and anterior cingulate gyrus according to a study by Ghashghaei et al. (2007), with implications involving focus on motivationally relevant stimuli. Studies have also shown less activity in the left side of the ventrolateral prefrontal cortex, specifically in automatic emotion regulation involved regions, throughout mania and remis sion in bipolar adults. Also, significantly reduced activity within the left sides of the dorsomedial prefrontal cortex and the orbitofrontal cortex throughout automatic attention and emotion regulation in bipolar adults. Overall, the neurodevelopment of bipolar disorder is quite obviously complex, but it is evident that abnormalities in regions of the left side prefrontal cortex as well as the left side hippocampus and parahippocampus, at least in childhood bipolar disorder, are present and should be further examined. (Phillips, 2008). According to Craddock et al. (2010) after analysis of a Wellcome Trust Case Control Consortium study, a gene that encodes the GABA receptor ÃŽÂ ²1 subunit, GABRB1 contained a significantly associated polymorphism, based on an analysis of bipolar patients and controls. After further studies they determined that variation within the GABA receptor genes can contribute towards risk of bipolar disorder. Therefore, this is yet another implication of a neurologically related issue in bipolar disorder patients, this time concerning a factor that could be related to alcohol issues and things of that nature in those with bipolar disorder (Craddock, 2010). According to Yuan-Hwa et al. (2010), midbrain binding of serotonin transporter (SERT) has been shown to be decreased in those in the depressive state of bipolar disorder in previous positron emission tomography studies. In the study completed by Yuan-Hwa et al., the goal was to analyze if the same dysfunction would apply to those in the euthymic state of the disorder. The study was conducted by using two rating systems, the Montgomery-Asberg Depression Rating Scale and Young Mania Rating Scale, with a less than ten score or less than seven score, respectively, over an eight week period classifying a euthymic state. To measure the midbrain binding of serotonin transporter, Yuan-Hwa et al. used single photon emission computed tomography and the radiotracer 123I-ADAM. Representing the availability serotonin transporter binding within the midbrain, the main outcome measured was speci ¬Ã‚ c uptake ratio (SUR) (Yuan-Hwa, 2010). Results included dramatically lower averaged specific uptake ratios for bipolar I patients than for bipolar II patients or healthy individuals according to Yuan-Hwa et al. (2010). There was also a strong correlation of decreased specific uptake ratios in bipolar I patients with illness duration. This means that there is a different biological regulation mechanism in bipolar I patients than in bipolar II patients following stable treatment, which could make a case for dichotomy in bipolar disorder (Yuan-Hwa, 2010). Overall, neurotransmitters and other neurological system factors play a large role in understanding bipolar disorder of various types and could potentially lead to diagnosis and treatment of the illness earlier rather than later, when it has the best chance at being successful. If one thing is clear related to the neurological piece of this bipolar puzzle, it is that abnormalities and decreased levels of functioning are present, and need to be further understood if progress toward better management of this disorder is going to take place. Miscellaneous Factors There are many other various factors that have biological implications in bipolar disorder. In this section, they will be discussed one by one. From protective factors to biomarkers that may increase risk, they will be explored in detail. The first factor concerns a certain kainate-type ionotropic glutamate receptor that is part of the glutamate neurotransmission process, called GRIK4, according to Pickard et al. (2006). Regarding bipolar disorder, a two single nucleotide polymorphism haplotype of the gene proved to be the most significant. Results regarding the haplotype inferred a protective quality towards bipolar disorder. Also, Pickard et al. identified multiple individual haplotypes from markers four to eight, which subsequently associated with a protective quality regarding bipolar disorder, even though that region did not appear within the global analysis (Pickard, 2006). Therefore, it is clear that this gene has properties which protect the individual from developing bipolar disorder. Regarding the reelin gene (RELN), which helps in brain function and development, Ovadia and Shifman (2011) have found that there is a link between the dysfunction of the gene and psychiatric disorders like bipolar disorder. The expression of the reelin gene and its various isoforms was studied using brain samples from postmortem patients of bipolar disorder and schizophrenia. Results indicated that there was a discernible reduction in the short reelin gene isoform proportion, which was lacking the C-terminal region, in bipolar disorder. Hence, the RELN, or more specifically its short isoform, is dysfunctional and that dysfunction can be associated with bipolar disorder (Ovadia and Shifman, 2011). Patterson et al. (2009) found a biological marker of bipolar disorder using sensory gating, and specifically the P85 gating ratio. Their goal was to discover if and how gating of an auditory brain potential at 85 ms (P85), which hadnt been previously tested, di ¬Ã¢â€š ¬erentiated patient groups and control groups. Patterson et al. collected P85 and P50 auditory evoked potentials from schizoa ¬Ã¢â€š ¬ective disorder patients, paranoid schizophrenia patients, and bipolar I disorder patients, and also from healthy control individuals. The results showed that the P85 gating ratio was dramatically greater in the bipolar group compared to any of the other groups; and the P50 gating ratio was dramatically greater in the schizoa ¬Ã¢â€š ¬ective group than it was in the control group, however it didnt di ¬Ã¢â€š ¬er from the ratio for the paranoid schizophrenia group or the bipolar group. Therefore, given the results, the P85 gating ratio might be a unique new biomarker for bipolar diso rder (Patterson, 2009). BD is associated with a number of genetic and possibly epigenetic abnormalities associated with neurotransmitter, hormonal and immunologically mediated neurobiological pathwaysà ¢Ã¢â€š ¬Ã‚ ¦HPA axis and immune system abnormalitiesà ¢Ã¢â€š ¬Ã‚ ¦ (Duffy, 2012). This study completed by Duffy et al. basically reviews various biological indicators pertaining to susceptibility to bipolar disorder. Certain studies examined differences in the hypothalamic-pituitary-adrenal (HPA) axis and also the immune systems of bipolar patients. In addition, there were implications to bipolar disorder found involving signal transduction processes within cells and also disruption within energy metabolism. Individuals at genetic risk provide a context for neurobiological findings and models concerning the onset and the progression of the illness are discussed. Overall, neuronal systems are dysfunctional in some ways in those with bipolar disorder, and because of this, things such as apoptosis concerning t he strain of the disorder and oxidative stress can occur, which obviously are not normal or healthy in any way (Duffy, 2012). Going in a little different direction, Kapczinski et al. (2009) studied recurrence of manic and depressive episodes and how it can contribute to the progression of the disorder. Neurobiological abnormalities may increase as episodes happen over and over, and as the number of episodes an individual has had increases, both treatment by medicine and by psychotherapy become less and less successful. This may be due to worsening dysfunction regarding insight, cognition, and relationships, and various other types of functioning. Also, the degree to which the illness has progressed can be assessed by the continuation of dysfunctioning throughout the fairly normal mood phases that bipolar patients experience (Kapczinski, 2009). Various indicators for bipolar disorder include, à ¢Ã¢â€š ¬Ã‚ ¦abnormalities in some biomarkers, such as brain derived neurotrophic factor (BDNF) and cytokines such as tumour necrosis factor alpha (TNF-alpha), which may be related to neuronal and glial dysfunction (Kapczinski, 2009). These specifically are relevant to the possible staging of the disorder as increased levels of TNF-alpha and decreased serum levels of BDNF characterize patients in the later stages of the illness when examined in relation to those in the earlier stages. Therefore, if studies are replicated and more evidence supports differences significant enough to classify stages of bipolar disorder with unique treatment options and things of that nature, patients may not need to suffer unnecessarily and more success can be achieved towards the goal of relatively normal lives for these individuals (Kapczinski, 2009). Conclusion To conclude, there is much that has yet to be discovered regarding the causes and various indicators, biological and otherwise, of bipolar disorder. However, progress is most definitely being made, and there are many promising avenues to pursue in terms of achieving a greater understanding of this illness and how it is developed. From genetics and neurotransmitters which clearly play a significant role, to circadian rhythm factors which seem to be quite valid, this disorder has many influences. While it may seem at first to be a disorder triggered by environmental factors and various life events, it really is heavily biologically incorporated into those who suffer from this life altering and sometimes uncontrollable illness. As more studies are completed that test for various abnormalities, dysfunctions, and possibly even new types of differences within individuals who have bipolar disorder and those likely prone to developing it, more factors will be discovered and it is more than likely that a better grasp on the complexities of the illness are not too far away. To put it simply, though patients of this disorder may feel hopeless and beyond frustrated at times, researchers do not, and the day when the illness is entirely understood and treatment options are consistently effective will come sooner than some people might think.

Employees’ Issues in Hcl Axon Malaysia

ORGANIZATIONAL ISSUES AND PROBLEMS Peripherally, HCL AXON is an excellence organization with increasing sophistication of its information technologies and offer huge scope of business benefits. HCL’s engaging in information systems projects and timely subjects helps yielding substantial achievement and continue to be robust. However, HCL AXON’s branch in Malaysia, HCL AXON Malaysia is actually having a big trouble in human resource management. HCL AXON Malaysia today fall prey to the same basic problems that were apparent all over the years in the organization.Questions have to ask whether organization is learning. Questions must also be asked about the quality of training given to project managers, developers as well as the IT support engineer. The employees are bewildered in a suffocating quandary which consists of rigidity in the Human Resource department, negligence of employees’ welfare, and the lack of employee development. i. Rigidity in the Human Resource department The HR department shows negligence in updating its information system. For instance, the working contract between a computer technician, Morty with HCL AXON Malaysia is expired in February 2012.Without contract extension, he is still working in the organization and being paid his salary every month until today. In fact, this situation is insecure to Morty because no one guarantees the termination would not descend one day against him. Furthermore, there is another similar example. A former employee, Tom quitted his job from HCL AXON Malaysia in September 2012. However, he was still being paid his salary in the next two consecutive months. After being informed about this case, only the HR department rectified the system.These might caused by the lack of initiation in the HR department towards developing deep awareness for contracting, staffing and recruitment system. They keep running the existing system like doing a daily routine, paying the fixed amount of wages to empl oyees on the list without any updating. Subsequencely, the HR system is not developing in fully aligned system which includes aligning the organization’s policies and human systems i. e. staffing. ii. Negligence of Employees’ Welfare Besides, the HR department in HCL AXON Malaysia never takes employees’ elfare and needs seriously. For instance, the IT engineers or computer technicians are usually work overtime and carrying out work-shift every week. The employees are also ought to work on public holiday. However, there are no allowance and bonus given. Employees crave for the benefits and compensation system would be ameliorated, but their request and allegation are being ignored by HR department. Meaning by that, the HR department shows laziness to get input or feedback from employees to improve the exacting situation.An IT engineer in HCL AXON Malaysia, Terence claims that the employees in the organization are mostly having low job satisfaction. iii. The Lack of Employee Development The changing environment and technologies such as launching of new software and IT systems as well as the unpredictable issues regarding foreign countries’ client system urge HCL AXON to utilize system approach to make continuous-improvements. The HR department of HCL AXON Malaysia promised to conduct training sessions to the employees which is training of using new software. However, the managers and supervisors never work diligently to meet the commitment.

Essay on “The Red Scarf Girl” by Ji Li Jiang

â€Å"The Red Scarf Girl† â€Å"Many friends have asked me why, after all I went through, I did not hate Chairman Mao and the Cultural Revolution in those years. The answer is simple: we were all brainwashed. †p. 276 The book â€Å"The Red Scarf Girl† is a memoir written by author Ji Li Jiang recounting what it was like to grow up during the Great Proletarian Cultural Revolution, taking place in China from 1965 to 1968.During this time, a political leader named Mao Zedong convinced the people of China that the â€Å"four olds†, or the old ways of China, were wrong and that the only way for their country to move forward was to completely revamp their beliefs and ways of life; basically creating a whole new culture for the Chinese. By ways of torture as well as basically brainwashing the people into thinking whatever he said was true, Chairman Mao single handedly shaped China’s culture by removing all anti-communist beliefs.During the first part of th is book, little things that Ji Li Jiang witnesses and says hints that Chairman Mao was forcing change onto the people of China. For example, in chapter 2, Ji Li helps destroy a sign for the Great Prosperity Market, saying that names like this are four olds. Many other words and phrases such as â€Å"fortune† and â€Å"innocent† were also considered four olds, and were not to be used. But this was just the first step of Chairman Mao’s plan.He also convinced his workers, called â€Å"the red guards†, to publicly humiliate people for various anti-Communist acts. These public humiliations got more and more violent as time went on; in the beginning of the Cultural Revolution, people were mainly humiliated for their clothing. On page 30, a man’s clothing is cut apart while he is standing in the middle of a street because the tight pants and pointed shoes he wore were considered four olds; â€Å"†¦tight pants and pointed shoes are what the Western b ourgeoisie admire.For us proletarians, they are neither good looking nor comfortable† said the Red Guard preforming this act. But as the story progresses, people are forced to wear dunce caps and signs with horrible messages written about them around their neck. They were then paraded around while people yelled horribly mean things at them, sometimes even throwing things. Many people were treated this way because of their, or someone in their family’s occupation; if you or anyone in your family was a landlord, you would definitely be treated this way.Landlords were believed to be â€Å"black welps†, meaning that they were evil people, because they would take land from the poor if they did not pay their taxes, and also often were wealthy enough to afford housekeepers. Many teachers were also treated harshly if the Red Guards had any reason to believe that they were teaching their students anti-communist ideas. The people of China did not think twice about this beh avior, however, because Chairman Mao had convinced them that people like this should be treated harshly. To us Chairman Mao was god. He controlled everything we read, everything we heard, and everything we learned in school. We believed everything he said. Naturally, we knew only good things about Chairman Mao and The Cultural Revolution. Anything bad had to be the fault of others. Mao was blameless. † (p. 276) Certain people were treated even more severely. Struggle meetings (gatherings within the workplace where people were publicly humiliated) were very common and often used physical violence to correct people.During one struggle meeting, a woman is beaten and forced to climb the chimney of a factory as a punishment, for she was believed to be a teacher pushing anti-communist beliefs on her students. Ji Li Jiang’s father is forced to partake in these daily because he was believed to be conspiring against the communist party. Even if The Red Guards did not have substa ntial evidence to torture these people, they had full permission to anyway under the power of Chairman Mao. â€Å"When I started to write this book, I asked An Yi’s mother if she had hated Mao when she was forced to climb the factory chimney. I didn’t hate him’ she told me. ‘I believed that the Cultural Revolution was necessary to prevent revisionism and capitalism from taking over China. I knew that I was wronged, but mistakes happen under any system. If the country was better for the movement that persecuted me, I was still in favor of it. It was only after Mao’s death that I knew I was deceived. † For Ji Li Jiang, it was not until her father was tortured that she realized that the Cultural Revolution may not have been great as it sounded. Before then, Ji Li Jiang actively participated in these events.So did all of her siblings and classmates. They all believed whole heartedly that Chairman Mao was doing this for the good of the people. Bef ore the Cultural Revolution, people had big ceremonies for their ancestors, and for the Chinese New Year. They believed in many superstitions, such as not sweeping the floor on New Year’s Day as to not sweep out the god of wealth, and eating spring rolls to â€Å"roll in the money†. People were not ridiculed because they were wealthy; wealth was admired and people strived for it. People collected stamps and other valuable things and cherished them with all their hearts.They kept photographs of themselves and their family members. They wore fancy clothes on special occasions, and the elderly kept elaborately decorated trunks containing all their families heirlooms and passed them down from generation to generation. People were allowed to hire housekeepers to help them around the house and with taking care of their children. But during the Cultural Revolution, none of these things were allowed. They were all considered four olds, and people’s houses were searched and pillaged to make sure that none of these things were going on.Years after the Cultural Revolution, Chairman Mao was proven guilty of unleashing this chaos onto the people of China purely to protect his own political position. â€Å"It was only after Mao’s death in 1976 that people woke up. We finally learned that the whole Cultural Revolution had been part of a power struggle at the highest levels of the Party. Our leader had taken advantage of our trust and loyalty to manipulate the whole country. This is the most frightening lesson of the Cultural Revolution: without a sound legal system, a small group or even a single person can take control of an entire country. This is as true now as it was then. †

The Effects of a Lack of Sleep - 734 Words

Insomnia; is also known as a deficiency of sleep or sleep deprivation. People with this condition are apt to suffer from inadequate sleep and more likely to battle depression, have poor concentration, and be involved in an auto accident; from a lack of focus. These issues followed by prolonged periods of time; can cause a person some sort of health issues, whether mental or physical.(Harvard Medical School) Sleep deprivation alters the production and action of some hormones, dampening the secretion of thyroid stimulating hormone and increasing blood levels, this shows mostly during the afternoon and evening hours. Higher cortisol levels are distinctive to much older subjects and are thought to be linked to age-related health problems, such as insulin resistance and memory complications. (University of Chicago) Associated things that cause insomnia that are common is stress, environmental changes, caffeine, cold medicine, steroids, and antidepressants. Some people deal with insomnia for only a few nights like jet lag, but if the problem persists it can progress into chronic insomnia, which is another common symptom of psychiatric disorders, such as; anxiety and depression. If sleep is interrupted then a balance of hormones can take place. As an example; poor sleep is the beginning to an increase in the production of cortisol, which is a stress hormone. This hormone affects the heart and too much of it causes hypertension or it can zap a person’s energy and bring them intoShow MoreRelatedThe Effects of a Lack of Sleep Essay1334 Words   |  6 PagesEffects of a lack of sleep, known as insomnia, is the most common classification of sleep disorders. It was estimated that thirty to forty million Americans have difficulty staying asleep, falling asleep or waking earlier than they would like to, and cannot get back to sleep. 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